About Joyce Holmes

Joyce Holmes lives with her husband and two small dogs in the beautiful Okanagan region of British Columbia, Canada. The greatest of pleasures can be found in the simplest of things. Playing with puppies, reading, and spending time with family, especially the grandkids. Joyce enjoys photography and blogging about her travels, and when she’s not planning her next adventure, she’s off enjoying life’s simple pleasures.

Living With Impaired Vision – No Wallowing Allowed

This is the final post about my eye health issues. I haven’t written these articles to garner sympathy. Rather, I hope to educate people to the symptoms and dangers of torn and detached retinas. And to let people who are going through a similar situation know that they aren’t alone. This is what happened, this is what was done about it, and this is what I’m left with. Maybe it’ll help.

My journey started in December 2016, with a natural occurrence that happens to most middle-aged people. As our eyes age, the vitreous gel that fills the central eye cavity liquefies and separates from the retina. When this happens, a person often sees floaters—dots, spots or curly lines, which follow eye movement. Usually this quickly settles down and vision returns to normal. Sometimes, the retina will tear or detach when the gel separates.

If you experience the above symptoms, seek medical care immediately. Call your optometrist without delay; there’s usually one on-call after hours. They’ll check for a break (tear) or detachment and refer you for any necessary procedures. A retinal detachment is an ophthalmologic emergency. The longer the wait for surgery, the lower the chances of a positive outcome. This can not only lead to irreversible vision loss, the health of the entire eye is endangered.

In my case, multiple breaks developed in my left eye’s retina, which I’ve had repaired twice with laser surgery. I now have large, mostly opaque, floaters in my eye that sometimes impede my vision. Rapid blinking usually improves things.

When the same episode happened to my right eye, in April 2017, it resulted in a vitreous hemorrhage. Blood vessels ruptured when the vitreous gel separated from the retina. This hemorrhage caused massive damage to my retina, completely detaching it.

I underwent a complicated surgery to repair my retina, involving a vitrectomy, scleral buckling, and injecting silicon oil into the eye cavity. I gave a detailed explanation of the procedures here.

Although my eye eventually healed, my vision was impaired, and I developed intraocular hypertension – high pressure in my eye. I’d hoped this would improve after the silicon oil was surgically removed in August 2017, but it didn’t.

Not only did the intraocular hypertension continue, I developed a cataract, and what little vision I had began to degrade. I had cataract surgery in July 2018, and while the surgery was successful, it didn’t improve my vision. This was when I first heard the words glaucoma and optic nerve damage in reference to my situation.

Like intraocular hypertension, glaucoma is too much pressure inside the eye cavity. It’s most common in older adults. Fluid in the eye cavity can’t drain properly, and over time the resulting high pressure damages the optic nerve. Early stage glaucoma has no symptoms because damage occurs gradually. In the later stages, symptoms include loss of peripheral vision, eye pain, and blurred vision. Vision damage is permanent and, if untreated, leads to blindness. There’s no cure for optic nerve damage, and various treatments may only prevent further vision loss.

Because vision loss can’t be recovered, it’s important to have regular eye exams that include eye pressure measurements. Seek immediate medical care if you experience sudden vision loss, halos around lights, any vision distortion or severe eye pain. These symptoms could result in optic nerve damage and permanent vision impairment.

In September 2018, I underwent a procedure called laser trabeculoplasty, which sometimes helps reduce intraocular pressure. It didn’t improve my situation.

In December 2018, I had an Ahmed valve implanted in my eye. Although the valve is functioning properly, my intraocular pressure remains unstable. I won’t know for several months whether the surgery will lower the pressure. Meanwhile, I continue to take Latanoprost with Timolol and Brimonidine eye drops.

So after two and a half years, and all those procedures, this is what I’m left with:

I have no functional vision in my right eye, and this is permanent. It’s like I’m looking through a lovely hoar-frost coated window, which is heavily frosted on the nasal side and less on the outer periphery, with the occasional clear spot where I have fleeting slivers of vision. With both eyes open, I see a general blurriness overlaid with an intricate pattern of lines. I often see a fainter version of this when just looking through my good eye.

When I close my eyes, or am in complete darkness, a lightshow starts up. Like looking through a black and white kaleidoscope. It’s elaborate and pretty.

I often have double vision through both eyes. The secondary image always cants to the upper left. It’s particularly bad while using the computer, and brings on tension headaches.

My right pupil is non-reactive and I’m extremely light sensitive, not only to sunlight, but also to bright overhead lights. I wear my pirate glasses, with a dark right lens, when shopping.

I lack depth-perception, so I bump into things, trip over things, and spill things. Everything requires greater concentration.

I’m no longer safe to drive, and I really miss this. So I’ve started using transit. It’s tediously slow, but oh well, it gives me independence.

My right eye aligns slightly to the right instead of looking straight ahead, and my eyelid sometimes droops – usually in bright light or if my eye is strained. This wide-eyed photo shows my dilated pupil, the Ahmed valve, the crooked eye and droopy eyelid.

Usually the valve is a barely discernible bump.

Recently, my right eye becomes increasingly red and irritated throughout each day. My ophthalmologist believes it’s extreme dry eye, made worse by computer-time. Hopefully using a lubricating gel, and patching my eye when on the computer for long durations will help. I swear my eye has a grudge against me, lol.

Despite all this, I don’t ask, ‘why me?’ Naturally, I wish this had never happened, but I don’t dwell on it. I absolutely know I’m lucky compared to the struggles of so many others. Of course I have bad days, but I never feel sorry for myself. It’s more frustration, impatience. I’ve always been a fairly competent person with high standards. I still have those high standards, I’m just not as competent, hence the frustration.

I also won’t play the ‘if only’ game. And I refuse to engage with anyone who wants to focus on the negative. Yes, this happened. Yes, perhaps the outcome should’ve been better. But, this is the outcome I’ve been dealt. Being negative won’t change anything, except make me miserable and bitter. I prefer being positive. I need to be happy, so I choose to accept my situation, and adjust to it the best I can, with no pity-parties.

To anyone dealing with eye problems, I wish you good luck. If you care to read through my articles, I’ve tried to give as much detail and information as possible. Click here to start.

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Recovering From Ahmed Valve Implant Surgery

I’ve been talking recently about the ongoing complications from a vitreous hemorrhage in my right eye, in April 2017. Catch up with current posts here, or start at the beginning of my journey here. These earlier articles contain pertinent information about retinal detachments and the serious problems that can arise.

Last week I described my Ahmed valve implant surgery. It takes eight full weeks to recover from this surgery, although the first two-three weeks are certainly the toughest. And rushing the process could put its success in peril, so don’t risk it.

For two weeks after surgery, do nothing that might exert pressure on the eye. No lifting of any type, gentle walking only, no housework, and no sexual activity. It’s even suggested to avoid getting constipated. So, seriously, no exertion.

It’s also important not to lean forward or tilt your head down. I had to sleep on my back or non-surgical side, propped up by several pillows for four weeks. I wore my eye shield to bed for a month, as well, as it’s crucial not to rub the eye.

Immediately after surgery, there was minimal swelling and redness. My eyelid drooped quite a bit, which is a protective response.

Day two:

Day four, redness quite apparent around the surgery site:

I saw my ophthalmologist one week after surgery. My ocular pressure was down to 9, which is low. He instructed me to cut back my eye drops to Latanoprost with Timolol, once a day.

Day nine, not much had changed.

The valve (a white rectangle) is barely visible under the inflamation:

I couldn’t bake goodies or decorate my house for Christmas—I didn’t even have a tree. I made the best of it, but my Christmas spirit was definitely lacking.

A pressure check, seventeen days post-op, came out a perfect 15 in both eyes. My eyelid was still droopy and swollen, and my eye continued to be quite uncomfortable. It sometimes felt itchy, sometimes gritty. The constant sharp pain was diminishing, but it still woke me up at night and throbbed badly if I leaned forward.

At four weeks post-op, it’s okay to start activities requiring light exertion. No heavy lifting or doing anything strenuous. If it affects your breathing, don’t do it.

At my four-week follow-up, my ocular pressure had elevated to 24. My ophthalmologist put me back on Brimonidine eye drops, twice a day. He thought the valve looked good:

My eyelid was still droopy. He explained that it’s protecting the eye, and as the eye heals, the eyelid position should improve. However, sometimes it remains lower than the other eye, especially if there’s been numerous surgeries, such as in my case.

With permission from the surgeon, we spent a week in Mexico one month after surgery. As a concession to my recovery, we slowed the pace of our long daily walks. Only once, climbing Mirador de La Cruz, did I feel serious eye pain and realized I was pushing it. A few minutes of rest and a gentler pace allowed me to carry on, pain-free.

At my six-week follow-up, my ocular pressure was 23, so hadn’t dropped despite increasing the eye drops. My ophthalmologist was concerned whether the valve was working properly, so faxed a letter to the surgeon in Vancouver. Sometimes a valve has to be readjusted surgically in the weeks following implant.

I flew to Vancouver two weeks later. Thankfully, my son came to my rescue again, chauffeuring me around and giving me his bedroom. At the appointment, the surgeon said my implant looked good and had healed well. My pressure was still 23, and he explained it might be unstable for up to a year, and I might always have to use eye drops. I told him the eye pain had started to fade around seven weeks post-op. He said that’s approximately when the stitches are completely dissolved.

A week later (two months post-op), the pressure reading was 24. I could now resume all activities, no matter how strenuous.

Three months post-op, the pressure reading was 25.

Four months post-op, the pressure reading was 17. Totally normal. It’s too soon to say if it’s stabilized, but I’m hopeful. I want to believe, after going through the ordeal of that surgery, that there’ll be one positive outcome in this long journey.

Click here for my final article, where I conclude my thoughts on my progress and go over some important information about eye health.

An Ahmed Valve Implant To Treat Glaucoma

I’ve been talking recently about the ongoing complications from a vitreous hemorrhage in my right eye, in April 2017. You can catch up with current posts here, or start at the beginning of my journey here. These earlier articles contain pertinent information about retinal detachments and the serious problems that can arise.

Last week I discussed stopping treatment for ocular hypertension and allowing my eye to go blind. After careful contemplation, I agreed to consult with a surgeon in Vancouver about having a shunt implanted. The consultation appointment was set for three weeks later, at the end of October 2018.

A friend agreed to stay with our puppies – the first time we’d ever left them. And our son kindly put us up at his apartment in Vancouver. The drive over the mountains was unexpectedly snowy.

We strolled around my son’s Mount Pleasant neighbourhood.

And enjoyed visiting him and his girlfriend.

A field vision test was performed at the appointment to confirm how much vision I still had. I looked into a dark tube and clicked a clicker every time I saw a light flash. First using my right eye, and then my left. I expected the surgeon to say I didn’t have enough vision to warrant doing the surgery. Instead, he strongly recommended it. He didn’t go into detail about the surgery, other than to say a tube would be placed in my eye cavity to help with fluid drainage. When I read the information sheets describing the surgery and recovery period, I realized how complex the entire procedure would be. Consequently, I left Vancouver still unsure whether to proceed or not.

At least the drive home was better. Clouds, but no snow.

I spoke with my ophthalmologist and family doctor, and they both felt I should have the surgery to preserve what little sight I still had in my right eye (which is important because my left eye is also visually and structurally compromised), as well as for the overall health of my eye. After considering this advice, I decided it’d be in my best interest to go forward with it.

Surgery was booked for December 11th, and I was told I’d have to stay in Vancouver for a week. We had to take the puppies with us, which meant we couldn’t stay at my son’s apartment. His girlfriend generously offered us her place, while she and her cat moved in with my son. I blogged about our stay, including photos, here.

The actual surgery was probably the most pain-free I’ve had so far. I asked not to be given a sedative, so the anesthesiologist didn’t even insert an IV. Instead of freezing my eye with needles (big ouch), a substance was instilled directly onto the eye (teeny, tiny ouch). This was reapplied several times during the procedure. The device implanted is called an Ahmed Valve. A tube goes from the eye cavity to a plate attached to the exterior white sclera of my eye, which then disperses the drained fluid. Pain was minimal throughout the surgery. Arrival to departure took two hours, surgery about forty-five minutes.

Throughout the day, my eye grew progressively more painful, and it leaked bloody tears nonstop, which, apparently, is not uncommon. By evening, the tears had turned clear, but were still profuse, also running inside my nose.

At my follow-up appointment the next morning, everything looked good and my ocular pressure was down (I don’t know the number). Fluid continued to leak, mostly into my nasal canal, and the eye was quite sore. If I accidentally tilted my head down (big no-no), the pain really ratcheted up.

The eye pain continued to worsen on the third day, although the leaking was diminishing. The following morning, I got the good news I could go home the next day. The bad news was that my ocular pressure had climbed to 28, which is high. The surgeon told me to expect fluctuations. As previously directed, I had stopped using the hypertension eye drops before surgery, and was now told to resume them.

The recovery period for this type of surgery is not easy. The slow, painful process took many long weeks, and I’ve detailed this in my next post here.

At a Crossroads—Go Forward or Give Up

I’ve been talking recently about everything I’ve gone through since suffering a vitreous hemorrhage in my right eye, in April 2017. You can catch up with current posts here, or start from the beginning here. These earlier articles contain some good information about retinal detachments and the various complications that can arise.

I ended my last post with the decision to try a laser treatment to help control my intraocular hypertension. I believe this procedure is called laser trabeculoplasty. The treatment, used primarily for open-angle glaucoma, can reduce the intraocular pressure by stimulating the eye cavity’s drainage system.

After having numbing drops instilled, the ophthalmologist (the same person who did my cataract surgery) placed a lens in front of my eye, and I saw bright flashes of light. The intense light beam assists in opening clogged tubes and draining eye fluid. This won’t cure hypertension, but helps control it. The procedure took minutes to complete, with only minor stinging pain.

When I described how I’d felt light-headed and weak since starting Methazolamide, he told me to stop taking the oral medication immediately. It obviously hadn’t been working anyway. He said I had uncontrolled acute open-angle glaucoma (which is the first time this term had been used to describe my condition) and I had two choices. Have the shunt surgery in Vancouver, or stop all treatment and allow my eye to go blind. Nothing else was working.

Once home, I educated myself about glaucoma. I learned:

– Glaucoma is too much pressure inside the eye cavity, like a basketball being overinflated. Fluid in the cavity can’t drain properly, and the resulting high pressure damages the optic nerve. This image shows my optic nerve already has some damage, which is impairing my vision. (center white spot on left-side orb)

– Early stage glaucoma has no symptoms because damage occurs gradually. In the later stages, symptoms include loss of peripheral vision, eye pain, and blurred vision. Vision damage is permanent and, if untreated, will lead to blindness.

– Glaucoma can occur at any age but is more common in older adults; it’s a leading cause of blindness in people over 60. Because vision loss can’t be recovered, it’s important to have regular eye exams that include eye pressure measurements. This way a diagnosis can be made early and treated appropriately. If glaucoma is recognized early, vision loss can be slowed or prevented.

I’m speculating that the silicon oil inserted with my original surgery contributed to my glaucoma. Not only can silicon oil in the eye cavity raise intraocular pressure, it can plug the drainage tubes, preventing them from functioning properly. These blockages could also hinder the effectiveness of the eye drops.

When I saw my regular ophthalmologist a few days later, I told him I’d decided to stop treating the hypertension. He was disappointed and voiced two concerns. Extremely high intraocular pressure can result in intense, unremitting eye pain. Also, once the eye is blind, it could ‘pop’, no longer moving with my other eye, and likely veering off to the right all the time. Both of these possibilities horrified to me.

I was about to leave on a week-long road trip and he convinced me to stay on Latanoprost, one of the drops I’d been taking, to prevent eye pain while away.

In the following two weeks, I felt quite conflicted. One day, I’d be at peace with my right eye going blind. Another day, the thought terrified me, especially with either of the two scenarios the ophthalmologist had described happening. So I made another appointment with my ophthalmologist.

My ocular pressure was 43, which is very high, but not surprising after having stopped most of my medication. I agreed to talk to the surgeon in Vancouver to find out more about the purposed surgery before making my final decision. He was so happy. He said to resume using all three eye drops, and he’d get a letter off to the surgeon immediately.

And I began another wait, cautiously optimistic. Not hoping to get my sight back, that possibility was behind me, but perhaps the health of my eye might finally stabilize. Click here for the next post.

Optic Nerve Damage – The Silent Vision Killer

Last week I described my cataract surgery and the disappointing outcome. I had set my hopes of better vision on that surgery, and although the procedure itself was successful, it did nothing to improve my sight. My eye problems started back in April 2017 after experiencing a vitreous hemorrhage. For those of you just joining this topic, you can follow my journey and find detailed information about retinal detachments here or you can catch up with my recent updates here.

When I saw my regular ophthalmologist a few weeks after cataract surgery, I asked what could be causing the worsening vision, if it wasn’t the cataract or the silicon oil residue. He said it was likely damage to the optic nerve from sustained elevated intraocular pressure. The cataract ophthalmologist had told me the silicon oil still in the eye cavity could raise ocular pressure. This ophthalmologist disagreed, saying the residue was too minimal to cause problems, although the oil could’ve originally plugged the drainage tubes.

My pressure was 18 that day, which is upper normal. In the past year it’d often reached the high 20s or 30s, and this can harm the optic nerve. He suggested I have regular pressure checks, and see him periodically. I didn’t know much about optic nerves, so did some online research. I learned:

-The retina and the optic nerve are two of the most important components of vision. The retina creates the image, and the optic nerve sends it to the brain for interpretation.

-Optic nerve damage can cause vision distortion, vision loss, and blindness. Seek immediate medical care if you experience sudden vision loss, halos around lights, any vision distortion or severe eye pain. These symptoms could result in optic nerve damage and permanent vision impairment.

-There are several causes of optic nerve damage, one being glaucoma or high pressure within the eye. Because glaucoma has no symptoms, optic nerve damage might not be detected until vision becomes impaired.

-There’s no cure for optic nerve damage, and various treatments may only prevent further vision loss.

When I had my pressure measured ten days later, it was up to 37. The ophthalmologist wasn’t in the office that day, but the tech contacted him, and he had her take an image of my eyes. He also prescribed an oral medication, Methazolamide, which eases pressure by slowing production of fluid in the eye.

I was already taking three sets of eye drops:

Latanoprost, which increases fluid drainage from the eye.

Timolol, which lowers the amount of fluid the eye makes.

Brimonidine, which helps with drainage. It also lessens the amount of fluid produced.

My blood pressure had been hovering in a lower than normal range, which the pharmacist said could be caused by the eye drops. At 108/59, it wasn’t a concern as long as I felt okay, which I did. Immediately after taking the Methazolamide pills, I began feeling dizzy and spacey, especially in the morning or after the slightest exertion. My blood pressure dropped to an average of 101/54.

Three days after starting Methazolamide, my intraocular pressure was up to 39. Three days after that it was down to 18. Ten days later, it was back up to 28. Despite all that medication, my eye pressure wouldn’t stabilize.

The ophthalmologist showed me the image of my eyes he’d had taken earlier that month. The orb on the left side (which is actually my right eye) reveals an increased whitening in the center, which indicates optic nerve damage. This damage is permanent, and would worsen if the intraocular hypertension isn’t controlled. The white mass on the upper right side is damage to the retina from the initial trauma.

There was an invasive surgery in Vancouver to consider, but first he wanted to try a simple laser procedure that’s often successful in lowering ocular pressure. The procedure could be done by the ophthalmologist who did my cataract surgery.

So I waited for that call. Meanwhile I crossed my fingers and hoped this procedure would work so I could stop taking that nasty Methazolamide.

And the saga continues. Click here for the next article.

The Results of Pinning My Hopes of Better Vision on Cataract Surgery

Last week I explained how my eye’s progress after a complex retinal detachment had mostly gone backwards in 2017. I faced the new year with worsening vision, a developing cataract and continued high ocular pressure. For those of you just joining this topic, you can find detailed information on the signs and dangers of retinal detachments, as well as the start of my journey, in my initial articles here, or catch up with my recent update here.

At my ophthalmologist appointment in mid-January 2018, I asked about having my eye cavity flushed again to remove more silicon oil residue, hoping this might improve my vision. He said cataract surgery was the priority, and he wouldn’t consider another flushing before then. He thought my eye looked good and he wouldn’t need to see me until after the cataract surgery.

When I got home, I realized my ocular pressure hadn’t been checked, nor had it come up in our conversation. Because I had an appointment at the end of that month with the ophthalmologist who’d be doing the cataract surgery, I decided to have him take a pressure reading. But not knowing was a worry, and I was disappointed it hadn’t been addressed that day.

At my appointment with the cataract ophthalmologist, I got the frustrating news of a probable nine-month wait for surgery. The ophthalmologist said my ocular pressure was elevated more than he liked, but he didn’t give a number. I told him I used Azarga eye drops twice a day to treat that. He didn’t have any further suggestions.

In April, I had measurement tests done, a routine procedure before cataract surgery. I didn’t see the ophthalmologist at that appointment. The blurriness and double vision were continuing to worsen, and I wondered if the cataract was causing this, and how bad it might become by the October surgery date.

In an unexpected turn of events, my cataract surgery was performed in July at the local hospital. It took approximately two hours, start to finish. Unlike my first two surgeries, applying the anesthetic didn’t involve inserting needles into my eye (yay!), and the surgery wasn’t painful. The surgeon kindly took the time to remove more silicon oil from my eye cavity during surgery. While my eye was only mildly uncomfortable afterward, I had a terrible headache and felt really off, so slept most of that afternoon. I blame this on the IV sedative given before surgery.

The next morning, the surgeon said the new lens looked great. Both he and I were surprised my sight hadn’t improved. He said it might still improve over time. He switched me from Azarga to Combigan eye drops to treat the elevated ocular pressure. I told him I’d just noticed my right eye was aligned slightly to the right instead of looking straight ahead. He said the surgery hadn’t caused this, and explained that when an eye doesn’t see well, the brain sometimes stops using it, causing it to wander off-center. This news, and the lack of progress with my vision, wasn’t encouraging. Ever the pragmatist, I decided to keep a positive mind-frame.

A week later, my ocular pressure had dropped slightly, so the cataract ophthalmologist had me continue with the Combigan drops and start another one, Latanoprost. Still no change in my eyesight.

Two weeks later, my ocular pressure was up to 37 (normal is 12-21), so the cataract ophthalmologist put me on a third eye drop, Brinzolamide. He said the gritty feeling in my eye was caused by dryness irritating my cornea, and suggested using lubricant drops. This was my last cataract check-up, and he forwarded his findings to my regular ophthalmologist, who I was scheduled to see in a few days.

So, a third surgery was behind me, and besides no longer having a cataract, my vision remained poor and unstable, and my ocular pressure remained high and untreatable. I had to face the stark reality of never regaining functional vision. My upcoming articles will address other challenges I had going forward. Click here for the next one.

The Aftermath of a Complex Retinal Detachment

Two years ago this month, my life changed forever when I awoke one morning with no vision in my right eye. Back then, I posted a series of articles on my journey going forward, and rather than repeat everything again, you can read them, starting here, if you’re interested.

Basically, I’d experienced what’s called a vitreous hemorrhage. As our eyes age, the clear vitreous gel that fills the central eye cavity liquefies and separates from the retina. When the gel separates, floaters may appear—dots, spots or curly lines, which move with the eye. This usually resolves quickly and vision returns to normal. Sometimes the retina tears or detaches when the gel separates. In my case, the eye hemorrhaged and the cavity filled with blood. The damage was enough to detach and severely damage my retina. This resulted in complicated surgery, including a vitrectomy, attaching a scleral buckle and inserting silicon oil.

My articles have been read upwards of a thousand times, and many people have reached out to compare stories. While I’m pleased to have offered reassurance or at least let them know they aren’t alone, a part of me is heartbroken that so many people have gone through this awful experience.

I stopped documenting my journey shortly before I had the silicon oil surgically removed. I wanted to have something constructive, some improvement, to share. So I kept waiting. I realize now, there won’t be a positive outcome for me. But it’s time to relay the rest of my cautionary tale, and reiterate once again to please take the best care of your eyes possible. It’s vitally important to recognize the symptoms and understand the dangers of torn or detached retinas. Don’t delay in getting medical attention if you have concerns.

At the end of August, 2017, I traveled to a nearby city, where the silicon oil was removed in a day-surgery procedure, which entails flushing the eye cavity numerous times to remove its contents. A local anesthetic was injected into my eye in two places, an intensely but fleetingly painful process. The actual flushing procedure only took about fifteen minutes. An air bubble was instilled into the eye cavity afterward.

Thankfully I didn’t have to do the awful head positioning that was required after my first surgery. There was minimal swelling and bruising around the eye compared to the previous surgery, and the pain was negligible. My eyeball was quite red and my vision was horrible, because of the air bubble. I had to patch my eye to read or watch TV. Walking was a challenge, too.

A week after surgery, the air bubble was only a third its original size. I had extreme and very clear nearsightedness, with anything more than a foot away just a blur. There were also lots of small black, very mobile, floaters. My ocular pressure was in the upper normal range, and I continued using an eye drop called Azarga, to control the high pressure.

Two weeks after surgery, the air bubble had completely dissipated. My distance vision was still nonexistent, beyond vague blurry shapes. My ophthalmologist had no suggestions for why my sight had worsen after the surgery. He said I was developing a small cataract that could be causing problems. Cataracts aren’t uncommon in situations like mine where numerous eye drops have been used over many months.

Four weeks after surgery, I still couldn’t see as well as before the oil was removed, but my vision had improved slightly. No change in the number of small black floaters. My eye pressure was beginning to climb back up above the normal range despite using Azarga.

Six weeks after surgery, my ophthalmologist conceded there may still be oil residue in my eye, causing the floaters and possibly affecting my vision. He said he’d consider flushing the eye cavity again if there was no further improvement. My eye pressure was up to 32, which is considerably high (normal is 12-21). Because I was already using Azarga to treat the condition, he opted to take a wait and see approach for a few months.

And that’s how 2017 ended for me. Oil removed, with possible residue remaining. Less vision than before I had the oil removed. A cataract developing, and the ocular pressure continuing to hover in the high range. None of this was cheery news, and I didn’t want to share my lack of positive progress for fear of discouraging others.

I’ve since had more surgeries and procedures, which I’ll explain in future posts, starting here.